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Fig. 1 | Alzheimer's Research & Therapy

Fig. 1

From: Plasma transferrin and hemopexin are associated with altered Aβ uptake and cognitive decline in Alzheimer’s disease pathology

Fig. 1

Modeling the association of plasma ceruloplasmin, transferrin, hemopexin, hemoglobin (Hb) α1, Hbβ, Hbδ, haptoglobin, haptoglobin-related protein (HPR), cluster of differentiation (CD) 163, and low-density lipoprotein receptor-related protein 1 (LRP1) with a brain amyloid deposition, b gray matter volume (GM Vol), c mini-mental state examination (MMSE), and d clinical dementia rating scale (CDR) score, in cognitively normal, mild cognitive impairment, and Alzheimer’s disease, in the AIBL cohort. The models included age, sex, APOEε4 genotype, and diagnosis as covariates. The standardized coefficient (β) and p values were stated: significant values that survive the false discovery rate correction set at 5% are highlighted in yellow

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Alzheimer's Research & Therapy

ISSN: 1758-9193